Diabetes / Metabolic Syndrome
The Blu Room provides a safe alternative to sun exposure and allows your body to make your own frequency specific vitamin D.
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The Relationship Between Vitamin D
and Insulin Resistance
Vitamin D is a steroid hormone that influences virtually every cell in your body, which is why maintaining a healthy level is so important. But vitamin D is equally important for your heart, brain, immune function and much more. For example, there's an important connection between insufficient vitamin D and both Type 1 and Type 2 diabetes.
Research showing this link includes but is not limited to the following:
Animal studies have shown vitamin D is a foundational factor necessary for normal insulin secretion and that vitamin D improves insulin sensitivity
One study involving nearly 5,680 individuals with impaired glucose tolerance showed that vitamin D supplementation increased insulin sensitivity by 54 percent
The mechanisms by which vitamin D reduces insulin resistance include its effect on calcium and phosphorus metabolism and by upregulating the insulin receptor gene
Incidence of several types of cancer could also be slashed in half, or more. Recent research reveals raising your serum 25(OH)D to 40 ng/ml can slash your risk of invasive cancers by 67 percent!
Most of the researchers specializing in vitamin D agree that sensible sun exposure is far better than vitamin D supplements which do not provide the same effects as the vitamin D your skin makes in response to UV exposure. However, chances are you're simply not getting enough sun exposure to raise your vitamin D level.
Ultraviolet Radiation Suppresses Obesity and Symptoms of Metabolic Syndrome
Read full article here
The role of vitamin D in curtailing the development of obesity and comorbidities such as the metabolic syndrome (MetS) and type 2 diabetes has received much attention recently. However, clinical trials have failed to conclusively demonstrate the benefits of vitamin D supplementation. In most studies, serum 25-hydroxyvitamin D [25(OH)D] decreases with increasing BMI above normal weight. These low 25(OH)D levels may also be a proxy for reduced exposure to sunlight-derived ultraviolet radiation (UVR). Here we investigate whether UVR and/or vitamin D supplementation modifies the development of obesity and type 2 diabetes in a murine model of obesity.
Long-term suberythemal and erythemal UVR significantly suppressed weight gain, glucose intolerance, insulin resistance, nonalcoholic fatty liver disease measures; and serum levels of fasting insulin, glucose, and cholesterol in C57BL/6 male mice fed a high-fat diet. However, many of the benefits of UVR were not reproduced by vitamin D supplementation. In further mechanistic studies, skin induction of the UVR-induced mediator nitric oxide (NO) reproduced many of the effects of UVR. These studies suggest that UVR (sunlight exposure) may be an effective means of suppressing the development of obesity and MetS, through mechanisms that are independent of vitamin D but dependent on other UVR-induced mediators such as NO.